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Figures 2, 4) availability is maintained by de novo synthesis from precursor amino acids, (glutamate, cysteine, and glycine), reduction of GSSG by glutathione reductase (GR), and uptake from exogenous GSH sources across plasma membranes (Figure 4) (62, 63)

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17,114 High-glucose and high-fat environments suppress SIRT1 expression, contributing to impaired insulin signaling and MASLD progression

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