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The effects of microbial-derived metabolites on kidney disease Evidence shows that SCFAs mitigate renal fibrosis by suppressing the expression of the pro-fibrotic chemokine monocyte chemoattractant protein-1 (MCP-1) induced by tumor necrosis factor- (TNF-) ( Figure 3 ) [88]

Network analysis revealed several metabolites exhibiting high connectivity, including S-adenosylhomocysteine, 5-methylthioadenosine, and diaminopimelic acid, reflecting their central roles within metabolic pathways rather than differential abundance between groups (Fig

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There are many possible causes