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[52] GPCRs respond to extracellular signals mediated by a huge diversity of agonists, ranging from proteins to biogenic amines to protons, but all transduce this signal via a mechanism of G-protein coupling

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GHK-Cu Research Mechanisms: What Studies Show Four mechanistic threads dominate the GHK-Cu literature: extracellular matrix synthesis, broad gene-expression modulation, wound-healing signalling via TGF- (Transforming Growth Factor-beta) and MMP (Matrix Metalloproteinase) pathways, and neuroprotection in rodent models