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Elucidating how this compartmentalized GPX4PRDX6 defense system suppresses early ferroptotic membrane damage remains an important future direction

Urbancsok, J., Bones, A

cerevisiae in which the ATP consuming glucose-glycogen bypass pathway had been shut-down by deletion of the GBE achieved 3.1-fold higher ATP-generating activity and 1.7-fold higher GSH productivity compared with the control strain [15]