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Conclusion The connection between genetic variants SNPs and diseases has revealed the importance of a metabolic pathway in the body that is critical to maintaining health, which is the methylation cycle

Conclusion In conclusion, we present the first intracellular proteomic analysis of CS exposure in fully differentiated phBECs, identifying NUPR1 as a candidate regulator of epithelial stress responses

An alternative strategy could involve the understanding of the molecular mechanisms underlying the known risk factors and to correct them, when possible, as a prevention/treatment of the disease

Upon activation of TAAR 1 , phosphorylation and activation of protein kinases A and C (PKA and PKC) occur due to intracellular cyclic adenosine monophosphate (cAMP) signalling [44, 45, 46]