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Absolutely yes

Together, these interconnected signaling cascades reshape transcriptional programs in fibroblasts, AECs, and endothelial cells, promoting myofibroblast differentiation, epithelial cell dysfunction, immune dysregulation, and excessive ECM deposition, which collectively drive the onset and progression of pulmonary fibrosis [33, 39] (Fig

Dimethyl fumarate (DMF), an FDA-approved drug prescribed for the treatment of multiple sclerosis ( Figure 7A ), allowing NRF2 to translocate to the nucleus and become transcriptionally active ( Figure 7 Indeed, our intracellular GSH detection assay revealed that both treatment with DMF and melatonin induced increases in ILC2 GSH levels ( Figure 7B )

doi: 10.1002/jsfa.8122 15 Gonzalez-RabadeNBadillo-CoronaJAAranda-BarradasJSOliver-Salvador MdelC