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Additionally, the development of pharmacological agents that precisely modulate intracellular iron homeostasis, such as iron chelators or iron oxide NPs, or enhance iron bioavailability specifically within the TME, may facilitate the targeted exploitation of ferroptosis pathways

Allerdings knnen wir der Zelle NAD-Vorstufen liefern, die dann im Zellinneren zu NAD umgebaut werden

However, the other two studies did not find significant associations (99, 109)

It also provides a new dimension to understanding immunopharmacology and immunotoxicology, with huge potential to impact disease etiology and pathogenesis, and for the development of novel and personalized therapeutics