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Use of the PARP-1 inhibitor, BMN673, reduced AIF and EndoG translocation after smoke exposure, confirming the role of PARP-1 activation in smoke induced activation of the parthanatos pathway in primary HBE cells

Quantitative analysis confirmed a substantial decrease in mitochondrial area and length, along with an increase in circularity in T-B2B compared with B2B (Fig

Smoke from burning wood is made of gases and fine particles which can be inhaled or ingested

ABOUT THE AUTHOR Ben Heskett Ben is a technology marketing executive, award-winning former journalist, lifelong music fan, and wine adventurer, always willing to try something new