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is appropriate for the protection of the public health because of, among several key considerations, the potential to reduce nicotine dependence in addicted adult smokers, who may also benefit from decreasing nicotine exposure and cigarette consumption

24 It has also been reported that treatment with LXR agonists can restore ABCA1 expression and reduce inflammatory responses following short-term cigarette smoke exposure, and in vitro treatment with LXR agonists can reduce macrophage inflammatory activation induced by cigarette smoke exposure

p-Cresol may contribute to atherosclerosis and thrombosis in patients with uremia and cresol intoxication possibly due to induction of ROS, endothelial/mononuclear cell damage and production of inflammation/atherosclerosis-related molecules (Chang et al

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