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This study provides evidence on how chronic inhalation exposure to aerosols from two alternative tobacco products impacts pulmonary inflammatory responses, induces oxidative stress, lung endothelial and epithelial damage, and suppresses the efficacy of vaccine-induced antibacterial immunity leading to increased pathogen burden in the lungs of exposed mice in a manner like combustible cigarette smoke (as graphically depicted in Fig

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PIR-1 exhibits ppp-RNA-specific binding activity In vitro studies on PIR-1 revealed a surprising activity associated with the presumptive catalytically dead C150S lesion

demonstrated that tobacco cigarette smoking was associated with a reduced flow-mediated dilation (FMD) [10]