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CONCLUSIONS The present study demonstrated that CSE may promote the apoptosis and senescence of EPCs by upregulating the expression of p300 and H4K12 protein, thus preventing the transition of EPCs from G1 phase to S phase, affecting telomerase synthesis, and reducing EPCs proliferation

In this context, the present article aims to explore current knowledge regarding the effects of radon and tobacco smoke on lung cancer, in order to identify possible pathways that could better model the synergies during co-exposure, while highlighting the gaps in our understanding of their interactions as combined mechanisms