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In inflammatory bowel disease, ATG16L1 has been shown to inhibit RIPK3-dependent necroptosis, thereby activating mitophagy, maintaining mitochondrial integrity, and reducing inflammation (82)

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The protocol of the current study was approved by the Ethics Committee of Jagiellonian University Medical College (decision number: KTEB/107/B/2000, project identification code: 6 P05D 112 20)

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