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P53 impairs the cystine/glutamate antiporter by silencing SLC7A11, its key component, resulting in cellular cysteine depletion and triggering ferroptosis

In summary, this article elaborates on the origins, extraction methods, and bioavailability of quercetin, emphasizing its specific mechanisms in intervening various cell death modalities (apoptosis, autophagy, necrosis, pyroptosis, ferroptosis, cuproptosis) and explores its specificity in action mechanisms from a physical organic chemistry perspective, especially concerning the liver

Boldfaced data indicate that the differences were statistically significant (Student's test 10 days) and not in planktonic cultures

Spreading of pre-existing segmental vitiligo after immunotherapy with house dust mite in a patient with atopic dermatitits