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As a key regulator of inflammatory cell death, ROS have recently been found to affect GSDMDs palmitoylation through various mechanisms

However, during a situation where massive apoptosis cannot be completed and converts into secondary necrosis with cell swelling and cell contents release, the extensive caspase activation of the primary apoptosis is still measurable [44]

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It is possible that the antioxidant activity of QCT reduces LPx and OS by regulating nitrites and nitrates, increasing glutathione (GSH), and decreasing MPO activity in the colonic mucosa (170) ( Supplementary Table 1 )