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MLH1 deficiency-triggered DNA hyper-excision by exonuclease 1 activates the cGAS-STING pathway

Furthermore, this research demonstrated that CU could degrade the levels of -syn by enhancing the autophagic function of nerve cells, suggesting that CU may mitigate oxidative stress-induced PD through multiple pathways

The optional addition of MOTS-C addresses the mitochondrial dysfunction that underlies menopausal metabolic slowdown

Perhaps the development and use of high-throughput screening technology and algorithm analysis tools are a viable strategy to promote the study of drug synergistic effects ( Presently, the mechanism of action of ruthenium complexes is unclear, and further research is still needed