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Our results suggest that the cell-autonomous control of GAB on CD4 + T cells is bimodal and consists of the sequential action of two processes, ABAT-dependent mitochondrial anaplerosis and the receptor-dependent signalling response, both of which are required for T cell-mediated inflammation

It upregulates IGF-1 expression in certain regions of the brain, such as the hypothalamus, cerebellum, and hippocampus

Lok et al., 2006

[7] References [edit] 1 2 3 GRCh38: Ensembl release 89: ENSG00000063854 Ensembl, May 2017 1 2 3 GRCm38: Ensembl release 89: ENSMUSG00000024158 Ensembl, May 2017 "Human PubMed Reference:"