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Even at extremely low copper concentrations, these cells experience severe copper overload, resulting in rapid ROS accumulation, collapse of the mitochondrial membrane potential, and disruption of the glutathione (GSH) redox system, ultimately activating various oxidative stress-mediated cell death pathways (e.g., apoptosis, necrosis, or cuproptosis) (205)

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Gamma-Glutamyl transpeptidase (gamma-GT) and maintenance of thiol pools in tumor cells resistant to alkylating agents

doi: 10.3390/polym9120720 210 DengQPanBAlamHBLiangYWuZLiuBet al