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Finally, a kavalactone analog, namely Compound 1, strongly inhibited LPS-stimulated iNOS induction and NO production, as well as Nrf2 signaling potentiation in vitro in microglial cells, highlighting the potential of kavalactone analogs to reduce neuroinflammation and maintain redox homeostasis [64]

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Although mucoadhesive artificial barriers enhance mucus concentration and retention, their potential interactions with MCC should be thoughtfully evaluated to ensure the preservation of the role of mucus in mediating water evaporation and assuring airway water homeostasis 1