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The Nuclear Receptor Rev-Erb Controls Circadian Thermogenic Plasticity

doi: 10.1089/ARS.2017.7083 63 CowleyH

Strong hydrogen bonds (type I) form a skeletal support structure between NCECS, Agarose, and PVA, while reversible hydrogen bonds (type II) contribute to dynamic self-repairing behavior, and the BPQD@MOF nanozymes are stably immobilized within the hydrogel network through type III interactions with PVA segments, which prevents nanozyme aggregation, leaching and structural collapse at the microscopic level, thus achieving the microstructural stabilization of the nanozymes

also thighs, upper arms) to avoid lipohypertrophy [14]