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2020), and it correlates poorly with a considerable fraction of parkinsonian symptoms

The density of these cells positively correlates with the severity of tau pathology, and their clustered aggregation is often a harbinger of neuritic plaque formation (149, 150)

Given this information, emerging therapies which target GSH homeostasis such as N-acetylcysteine (NAC), GSH esters, or activation of antioxidant response elements may provide new avenues to interrupt oxidant-driven inflammation in these diseases

This claim is supported by the identification of ZnT8, a protein responsible for Zn regulation that may be involved in the process leading to T1DM and T2DM [41, 42]