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If left unaddressed, a deficiency can result in severe complications, including irreversible nerve damage or memory impairment, making early diagnosis and ongoing treatment essential

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Similarly, pharmacological inhibition of HSL and knockdown of PNPLA2 reduced the expression of known PPAR target genes in which LDs were shown to be a source of PPAR and PPAR ligands that could be generated within minutes of stimulating lipolysis (3)