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In addition, NNMT knockdown upregulated tumor suppressor PP2A activity in GBM by increasing methyl availability for leucine carboxyl methyl transferase 1 (LCMT1) and increasing PP2A methylation while concomitantly decreasing the activity of serine/threonine kinases such as Akt and p38 MAPK (95)

Neither saturates the other's receptor system, allowing full effects from both agents simultaneously

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