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Enriching the LAT2 pathway in activated B cells and monocytes supports the involvement of B cell-mediated autoimmunity in endometriosis-associated infertility (89)

B., Nordborg, C

and controlled delivery systems that achieve and bind to targeted site via different stimuli or due to specific conditions (temperature and pH), followed by antibacterial activity while providing controlled cell/ion/growth factor release from the matrix (Jacob et al., 2018) (Figure 3)