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However, lack of quality control of the analysis makes these data uncertain

Therefore, inhibiting the polarization of macrophages towards M2 type and promoting the polarization towards M1 type may be beneficial for the treatment of GBMs (134)

What that next generation of disease modifiers might targetprotein aggregation, inflammation, cholesterol handling, metabolism, or another disease process altogetheris an open question

Given that N-acetyl-aspartyl-glutamate (NAAG) was recently identified as a glutamate reservoir 41 , we explored the possibility that metabolites containing glutamyl or glutamate groups could fuel glutamate synthesis in tumors