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Transcriptomics analysis of Sox9 mutant mice showed that deletion of Sox9 resulted in progressive disc degeneration and alterations in ECM organization indicating that Sox9 expression can also directly impact disc cell survival and ECM maintenance (Tsingas et al., 2020)

However, the absence of human clinical trials means the safety profile in humans is completely unknown

The focus here is on the peptide's ability to promote tissue repair and potentially calm the inflammatory response that often impacts the central nervous system in neurodivergent individuals

Wherever healing is already underway, it simultaneously reinforces 4 processes, angiogenesis (new vessels), cell migration (movement of cells to the site), proliferation (division) and the anti-inflammatory response