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In a rat model, our previous research on early NHPN-1010 treatment for blast-induced tinnitusspecifically targeting cochlear synaptopathydemonstrated that (1) blast overpressure caused a permanent loss of cochlear nerve ribbon synapses (RSs), and (2) administration of NHPN-1010 within 3 days of blast trauma, compared to placebo, significantly rescued RSs, as evidenced by ribbon counts in cochlear histological analyses and corresponding functional changes in suprathreshold wave-I amplitudes of the auditory brainstem response (ABR) 34

NSAIDs should be reserved for symptoms and inflammatory conditions for which lower risk analgesics (e.g., acetaminophen) have either failed or are not clinically indicated

A mechanism for epithelial-mesenchymal transition and anoikis resistance in breast cancer triggered by zinc channel ZIP6 and STAT3 (signal transducer and activator of transcription 3) Biochem