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Recent genetic and biochemical studies suggest a key role for adipose tissue in the development of insulin resistance, potentially by releasing lipids and other circulating factors that promote insulin resistance in other organs

The highest SE value observed in the CAA was 1.41 at the Ac-G combined dose of (1 + 0.125) g/mL, while the highest SE value observed in the cytoprotective effects was 1.61 at the Ac-G combined dose of (1 + 0.5) g/mL

Significant increases in macrophages and neutrophils [68], pro-inflammatory cytokines (TNF- and IL-1) and chemokines (CCL2 and CCL3) [69], cell apoptosis [4], oxidative stress (reactive oxygen species and reactive nitrogen species), and the suppression of antioxidant proteins (such as glutathione peroxidase, catalase, and superoxide dismutase) are all frequently linked to COPD-induced airway obstruction [70] in both the airways and lung parenchyma, in response to noxious particles and gases [71]

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