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Treatment with XPro1595, a selective inhibitor of solTNF that crosses the intact blood-brain barrier (BBB), in cuprizone-fed mice did not prevent toxin-induced oligodendrocyte loss and demyelination, but it permitted profound early remyelination due to improved phagocytosis of myelin debris by CNS macrophages and prevented disease-associated decline in motor performance

Each tube was labeled with the subjects ID, visit number, date, and time of collection

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The correlation between metabolite levels and scale scores in ASD group was evaluated using Spearmans correlation