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Neoalbaconol (NA), a compound derived from the fungus Albatrellus confluens, has been found to trigger necroptosis by facilitating the autocrine release of TNF through the modulation of the RIPK/NF-B signaling pathway and RIPK3-dependent reactive oxygen species (ROS) generation (Yu et al., 2015)

Subsequent serial echocardiographic evaluations were done during the Dox administration process on the following points: 14, 24, 37, and 52 days (Figure 1)