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FIN56, a ferroptosis inducer derived from CIL56, demonstrates greater selectivity for ferroptosis in comparison to its parent compound

[DOI] [PubMed] [Google Scholar] [119].Hughes VA, Fiatarone MA, Fielding RA, Kahn BB, Ferrara CM, Shepherd P, Fisher EC, Wolfe RR, Elahi D, Evans WJ

The inflammation-resolving role of n-3 PUFAs may also be supported by the fungal-derived extract, Albatrellus confluens , which is a source of the bioactive phenolic compounds grifolin and neogrifolin [27]