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These results suggest that the downregulation of GPx4 is critical for DOX-induced ferroptosis and that differential behaviors of GPx4 expression in response to DOX might be responsible for the susceptibility of DOX-induced injuries through ferroptosis

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Honda, Y., Kessoku, T., Sumida, Y., Kobayashi, T., Kato, T., Ogawa, Y., Tomeno, W., Imajo, K., Fujita, K., Yoneda, M., Kataoka, K., Taguri, M., Yamanaka, T., Seko, Y., Tanaka, S., Saito, S., Ono, M., Oeda, S., Eguchi, Y