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The decreased concentrations may be explained by the fact that the study participants were likely older than the subjects used to establish the laboratory norms

DNA damage by lipid peroxidation products: implications in cancer, inflammation and autoimmunity

[117] also investigated the impaired immune response with age in relation to carotenoid intake (-carotene, lycopene, lutein), finding that age-related changes in immune markers such as higher serum IgA levels and altered lymphocyte subpopulations (increase in memory Th cells (CD4 + CD45RO+) and natural killer (NK) cells along with a decrease in nave Th cells (CD4 + CD45RA+) and B lymphocytes) and impaired neutrophil activity, which could be modulated by carotenoid intake, depended only marginally on dietary carotenoid depletion and repletion [117]

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