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Recently, Nascimento and colleagues [111] analyzed the effect of CBD on nociception in experimental PD, highlighting the ability of this drug to exert antinociception via activation of CB1-R

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5A), we hypothesized that Cyss mediates protein stabilization of NRF2 and that KEAP1 is involved in sensing intracellular Cyss levels by direct cysteinylation of its sensor Cys residues