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other: n = 5), yielding 62 patients who completed 12-month follow-up by March 2025 (Table 1)

670,673 Aberrant epigenetic modifications within endothelial cells may further impair liver regeneration, 674 highlighting potential therapeutic targets to restore regenerative capacity in NASH

Aged HSCs exhibit reduced self-renewal and repopulation capacity, a myeloid bias, and an increase in their frequency, while proteostatic mechanisms are compromised concomitant with chronic low-grade inflammation 11,12,13

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