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Treatment with RY-A significantly improved these pathological changes and attenuated the CCH-induced neural tissue damage

24 These substances promote inflammation as well as sensitize nociceptive pathways, leading to increased pain sensitivity, known as hyperalgesia, and pain from normally non-painful stimuli, termed allodynia

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NO exhibits well-characterized pleiotropic biological activities, including potent vasodilation, anti-inflammatory actions, modulation of ECM metabolism, and suppression of apoptosis