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GO enrichment analysis showed that the upregulated DEGs were mainly enriched in apoptosis, inflammation, and oxidative stress, while the downregulated DEGs were primarily involved in gap junctions, cell proliferation, and sex hormone response (Figure 3B), suggesting that the damage to granulosa cell function, including increased apoptosis and decreased proliferation ability, may be a critical cause of follicular atresia and oocyte maturation disorders

The results showed that 2,4-D did not exhibit any agonist activity toward human estrogenic (hER) and androgenic (hAR) receptors across the entire range of tested concentrations (Figs

Alex Tatem, a board-certified urologist and member of Rugiet's Medical Advisory Board, will testify at this week's FDA hearings