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Upon starvation, CLUH inhibits mTORC1 activation and mitochondrial anabolic pathways, promoting mitochondrial turnover and allowing efficient metabolic reprogramming

Further study has shown that the chelator PBT2 forms a ternary Cu(II) complex with A that is highly stable but has low specificity (109)

To confirm that interface IV is essential for c-MET activation, we introduced Y673A mutation into HGF to disrupt this interface, and showed that HGF Y673 mutant has a great deficiency in binding to c-MET as well as promoting c-MET activation (Fig