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While glutathione may support liver health in some contexts, an unregulated high dose could place unnecessary strain on these organs if they are already compromised

Activation of the CaR-CSE/H2S pathway confers cardioprotection against ischemia-reperfusion injury

Interestingly, both of these sites are known to be important for NO binding and S -glutathionylation [14, 55], and because of the nature of Cys residues, a fine-tuned combination of multiple co- or post-translational modifications of these sites (including S -palmitoylation) could be important for regulating the multiple functions of GSTP1