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As mentioned previously, pancreatic -cells express TLRs, which can modulate -cell viability, influence insulin homeostasis (122), and contribute to T1D initiation upon interaction with high-mobility group box 1 (HMGB1) (123)

GHK-Cu provides a double-layered defense: Anti-Inflammatory: It reduces pro-inflammatory cytokines that trigger follicle "shutdown." Antioxidant: It neutralizes free radicals, protecting the follicle's DNA from environmental damage and aging

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In IL-10-deficient murine models, docosahexaenoic acid (DHA) mitigates CD symptoms by inhibiting the mTOR pathway and triggering autophagy [27]