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Results DMF activates the TSS pathway in SCD erythroblasts To investigate the mechanism of DMF on ferroptosis regulation, we conducted untargeted LC-MS using erythroblasts generated from CD34+ stem cells isolated from the peripheral blood of SCD patient

Cnrs, Inserm, Utcbs, France, (4)Fibronostics US Inc, USA Background: Chronic hepatitis B (CHB) and non-alcoholic fatty liver disease (NAFLD) are both common liver conditions

Additionally, although dietary iron overload did not increase the dystrophic pathology, total muscle iron content and ferritin expression were increased

Homogenates were centrifuged at 800 g for 10 min at 4 C, and the supernatant was sonicated on ice for 20 s before protein quantification (BCA protein assay, Pierce, Rockford, IL, USA)