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3436 This bacteria-induced NF-B activation and inflammatory protein expression was decreased by glutathione augmentation, suggesting a role for oxidants in COPD treatment

In response to this phase separation, research has developed icFSP1, which, by phase separating from FSP1 (a parallel pathway to GXP4), forms aggregates that inhibit the function of FSP1, reduce its affinity for membranes, decrease ubiquinone and vitamin K, and ultimately induce ferroptosis in tumor cells [101]

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Therefore, FXR/TGR5 dual agonist may exhibit additional effects in the treatment of DN (Wang X