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R.WangY.YuY.et al (2022b)

A cascade hypothesis The prevailing pathogenic hypothesis for AD is the amyloid cascade hypothesis, which claims that amyloid precursor protein (APP) is cleaved to produce A, leading to the aggregation of oligomers [23, 24]

It can be seen that VEGF is the main regulator of wound revascularization

Recent studies revealed that oxidative stress and mitochondrial dysfunction can be associated with its pathogenesis and a reduced rate of ATP synthesis has been reported [32,33,34]