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Oxygen-sensitive mitochondrial accumulation of cystathionine beta-synthase mediated by Lon protease

Evolving theories of the pathophysiology of major depression have implicated overactivation of inflammatory pathways, ill-defined changes in glutamate metabolism, and disconnection of cortical and subcortical components of a neural reward circuit

98 In the context of AMI, such modulation is protective in the short termreducing force output and protecting the ACL from further stressbut it risks persistent muscle inhibition, atrophy, and weakness over time

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