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These findings suggest that a strategy aimed at sustaining or enhancing brain GSH may be a viable approach to mitigate secondary injury processes induced by TBI

"N-acetylcysteine add-on treatment in refractory OCD." J Clin Psychopharmacol

Its five main mechanisms of action are as follows: (1) Increases the threshold for migraine attacks by activating different sites on the gamma-aminobutyric acid (GABA) receptors in brain neural tissue, decreasing neuronal excitability, boosting membrane hyperpolarization, and decreasing focal discharges

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