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Although the favourable effect of tirzepatide on this endpoint appeared to be predominantly driven by a reduction in new onset macroalbuminuria [22], it is worth noting that the differences in changes in albuminuria during the time course of the study were mainly attributable to a progressive increase in the insulin glargine group whereas levels remained relatively stable in the tirzepatide-treated group

In wound infections, TLR signaling is activated by PAMPs such as lipopolysaccharides (TLR4 ligands), lipoteichoic acid (TLR2 ligands), flagellin (TLR5 ligand), and unmethylated CpG DNA (TLR9 ligand) derived from invading bacteria, fungi, or viruses [32]

Contribution of cytokine-induced neutrophil chemoattractant CINC-2 and CINC-3 to neutrophil recruitment in lipopolysaccharide-induced inflammation in rats