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Conversely, the interaction between ROS and the NF-B pathway also involves the activation of antioxidant gene expression, which helps protect against ROS damage [223]

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These transfers were absent when TERRA12 rG4 was examined in isolation by saturating different imino peaks, indicating that the observed saturation arises from complex formation (Supplementary Fig

It would be beneficial if compound 102 can inhibit Hv1 at the same time while activating GPX4, as both lead to the reduction of ROS and neuroprotective function