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10.1002/jimd.12009 24 GiovinazzoD.BursacB.SbodioJ

Collectively, these results indicate that hippocampal astrocytic Hb, rather than CA1 pyramidal neuronal Hb, is necessary for the antioxidative action of KDS12025, effectively alleviating astrogliosis and hippocampus-dependent memory impairment in an animal model of AD

Antioxidants in the fight against atherosclerosis: is this a dead end

During metabolic stresses such as increased oxidative phosphorylation or changes in cytoplasmic homeostasis that induce ER stress, the UPS will be compromised, which would most likely result in a reduction in BACE1 degradation by the UPS and lysosomes