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As will be discussed, one mechanism which may connect thiol-metal binding to oxidative stress-mediated toxicity is specific of Hg, the cysteinyl-glycine bound form of which is able to deplete glutathione reservoirs by a catalytic process [34]

The lower number in testis resulted from the exclusion of homologous gene interference and our stringent focus on differentially expressed transcripts

Conclusion Multidrug resistance to chemotherapy drugs is one of the main obstacles in human cancer chemotherapy and has prompted intense research into discovering novel and innovative mechanisms that can overcome this barrier

Meanwhile the drug species, combination and delivery strategies of cuproptosis inducers or inhibitors also await further exploration