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However, the therapeutic use of cisplatin is limited by its irreversible side effects, including transcription inhibition, cell cycle arrest, generation of reactive oxygen species (ROS), and apoptosis [3]

The best example is the mechanisms for downregulating HMGB1, which we could deduce to be a therapeutic target for alleviating inflammatory reactions and thereby the associated DCM, particularly the myocardial damage evidenced by the rodent models utilized in the present study

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Role of catalase in oxidative stress -and age-associated degenerative diseases