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TMAO has been implicated in angiotensin II-mediated vasoconstriction (through enhancement of intracellular calcium release), increased sympathetic activation and oxidative stress, and impairment of endothelial nitric oxide production (involved in vasodilation) (Fig

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The BB-enhancement was shown to be mediated by BB1 attachment to the TLR-2 complex on the apical surface of enterocytes, leading to the activation of the p38 kinase signaling pathway and also the inhibition of NF-B